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Frontiers | Pathological role of methionine in the initiation and progression of biliary atresia

Four-week pretreatment with LA and/or ALC attenuated the rotenone-induced GSH reduction and oxidative protein and DNA damage Mitochondrial dysfunction is one of main sources of oxidative stress

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The immunofluorescence analysis of liver cross-sections also confirmed that the ethanol administration stimulated protein S-glutathionylation, while GSH significantly regulated protein S-glutathionylation (Fig

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